Short elements within a prion protein's sequence can cause it to activate and even cross the species barrier to spread neurodegenerative disorders such as Creutzfeldt-Jakob disease to humans
"One kind of pie in the sky idea is, if you can understand these regions within disease-related prions," says Tessier, "[you can] use this to identify small molecules and peptides that may be able to block how these things interact with each other and which are key for blocking this [prion] aggregation from happening."
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